The current study showed that b-asarone decreased the a-synuclein expression and played a role in protecting neurons from MPTP or MPP+ induced injury, suggesting b-asarone as a potential therapeutic agent for PD. In addition, MALAT1 was proposed to be a promising target molecule involving in the pathophysiological processes of PD.
NSDNA: The Nervous System Disease NcRNAome Atlas
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