A Manually Curated Knowledgebase of Aging across a variety of Species

Details


Name ASF1a
Type histone modification
Species Homo sapiens
Tissue liver
Experiment Method qPCR
Up/Down Up
Pro/Anti Pro
Funtion Description ASF1a overexpression is widespread in human malignancies and is required for the infinite proliferation of cancer cells, whereas its inhibition induces DNA damage and subsequent up-regulation of p53-p21cip1 expression, thereby triggering cellular senescen
Regulation Gene p53-p21cip1
Year 2019
Pubmed ID 30692519
Title ASF1a inhibition induces p53-dependent growth arrest and senescence of cancer cells.
Drug NA
Disease HCC
Environment Factors NA
Circulating NA
Variant NA
High-throughput NA