A Manually Curated Knowledgebase of Aging across a variety of Species

Details


Name Dot1l
Type histone modification
Species Mus musculus
Tissue bone
Experiment Method PCR
Up/Down differential expression
Pro/Anti NA
Funtion Description Both Dot1l-deficient strains exhibit a phenotype characterized by joint remodeling with extensive osteophyte formation and ectopic ossification upon ageing, indicating accelerated development of spontaneous osteoarthritis.
Regulation Gene DOT1L
Year 2019
Pubmed ID 30513362
Title Increased susceptibility to develop spontaneous and post-traumatic osteoarthritis in Dot1l-deficient mice.
Drug NA
Disease Osteoarthritis
Environment Factors NA
Circulating NA
Variant NA
High-throughput NA