A Manually Curated Knowledgebase of Aging across a variety of Species

Details


Name H2AX
Type histone modification
Species Homo sapiens
Tissue human neuroblastoma cells
Experiment Method qPCR
Up/Down NA
Pro/Anti Pro
Funtion Description We demonstrate that telomere dysfunction induced by adenovirus-mediated expression of dominant-negative TRF2 (DN-TRF2) triggers a DNA damage response involving the formation of nuclear foci containing phosphorylated histone H2AX and activated ATM in each
Regulation Gene H2AX
Year 2006
Pubmed ID 16539655
Title TRF2 dysfunction elicits DNA damage responses associated with senescence in proliferating neural cells and differentiation of neurons
Drug NA
Disease NA
Environment Factors NA
Circulating NA
Variant NA
High-throughput NA