Details
| Name | ATM |
| Type | gene |
| Species | Mus musculus |
| Tissue | MEFs |
| Experiment Method | other |
| Up/Down | NA |
| Pro/Anti | NA |
| Funtion Description | The ability of 53BP1 to promote the mobility of dysfunctional telomeres could explain why ATM kinase, its upstream regulator, is required for telomere fusions. Indeed, similar to 53BP1-deficient cells, dysfunctional telomeres failed to gain their maximal |
| Regulation Gene | NA |
| Year | 2008 |
| Pubmed ID | 18931659 |
| Title | 53BP1 promotes non-homologous end joining of telomeres by increasing chromatin mobility. |
| Drug | NA |
| Disease | NA |
| Environment Factors | NA |
| Circulating | NA |
| Variant | NA |
| High-throughput | NA |