A Manually Curated Knowledgebase of Aging across a variety of Species

Details


Name miR-34a
Type miRNA
Species Homo sapiens
Tissue Knee joint and caput femoris cartilage
Experiment Method RT-PCR,B-galactosidase staining,Luciferase reporter gene assay,Western blotting
Up/Down Up
Pro/Anti Pro
Funtion Description These findings indicate that miR-34a contributes to chondrocyte death, causing OA progression through DLL1 and modulation of the PI3K/AKT pathway.
Regulation Gene DLL1
Year 2018
Pubmed ID 30048987
Title MiR-34a Enhances Chondrocyte Apoptosis, Senescence and Facilitates Development of Osteoarthritis by Targeting DLL1 and Regulating PI3K/AKT Pathway.
Drug NA
Disease Osteoarthritis
Environment Factors NA
Circulating NA
Variant NA
High-throughput NA