A Manually Curated Knowledgebase of Aging across a variety of Species

Details


Name miR-34a
Type miRNA
Species Homo sapiens
Tissue lung
Experiment Method Real-time PCR,Western blotting
Up/Down Up
Pro/Anti Pro
Funtion Description In summary, we found that miR-34a upregulation in AECs of aged animals promoted dysfunctions of these cells by inducing cellular senescence, mitochondrial aberration, and cell death and thus leading to pulmonary fibrosis in the aged population.
Regulation Gene Sirt1,E2F3,E2
Year 2017
Pubmed ID 27979858
Title miR-34a promotes fibrosis in aged lungs by inducing alveolarepithelial dysfunctions
Drug NA
Disease fibrosis in aged lungs
Environment Factors NA
Circulating NA
Variant NA
High-throughput NA